ScienceDaily: Top Health News |
- Amniotic fluid stem cells repair gut damage
- Nuclear life of actin uncovered: Protein with key job in muscle function moonlights in nucleus to help regulate genes
- Parkinsons' drug helps older people to make decisions
- Molecular roots of Down syndrome unraveled
- DNA damage occurs as part of normal brain activity, scientists discover
- Molecular ‘signature’ for rapidly increasing form of esophageal cancer discovered
| Amniotic fluid stem cells repair gut damage Posted: 24 Mar 2013 05:20 PM PDT Stem cells taken from amniotic fluid were used to restore gut structure and function following intestinal damage in rodents, according to new research. The findings pave the way for a new form of cell therapy to reverse serious damage from inflammation in the intestines of babies.  |
| Posted: 24 Mar 2013 12:23 PM PDT Actin, a protein with a well-known job in the cytoplasm and an active role in muscle contraction, moonlights in the nucleus. It's function there was not understood, but now scientists find that it connects with chromatin.  |
| Parkinsons' drug helps older people to make decisions Posted: 24 Mar 2013 12:23 PM PDT A drug widely used to treat Parkinson's disease can help to reverse age-related impairments in decision making in some older people, a new study has shown.  |
| Molecular roots of Down syndrome unraveled Posted: 24 Mar 2013 12:23 PM PDT What is it about the extra chromosome inherited in Down syndrome -- chromosome 21 -- that alters brain and body development? Researchers have new evidence that points to the protein SNX27. SNX27 is inhibited by a molecule encoded on chromosome 21. The study shows that SNX27 is reduced in human Down syndrome brains. Restoring SNX27 in Down syndrome mice improves cognitive function and behavior.  |
| DNA damage occurs as part of normal brain activity, scientists discover Posted: 24 Mar 2013 12:22 PM PDT Scientists have discovered that a certain type of DNA damage long thought to be particularly detrimental to brain cells can actually be part of a regular, non-harmful process. The team further found that disruptions to this process occur in mouse models of Alzheimer's disease -- and identified two therapeutic strategies that reduce these disruptions.  |
| Molecular ‘signature’ for rapidly increasing form of esophageal cancer discovered Posted: 24 Mar 2013 12:18 PM PDT New research may offer clues to why rates of esophageal adenocarcinomas (EAC) have risen so sharply. The findings point to abnormal genes and proteins that may be lynchpins of EAC cell growth and therefore serve as targets for new therapies.  |
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